Esme murphy cancerous moles
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Publications U April
Arheix-Parras S, Barrios C, Python G, Cogne M, Sibon I, Engelhardt M, Dehail P, Cassoudesalle H, Moucheboeuf G, Glize B. A systematic review of repetitive transcranial magnetic stimulation in aphasia rehabilitation: leads for future studies. Neurosci Biobehav Rev. Apr 20;
Baldi I, De Graaf L, Bouvier G, Gruber A, Loiseau H, Meryet-Figuiere M, Rousseau S, Fabbro-Peray P, Lebailly P. Occupational exposure to pesticides and central nervous system tumors: results from the CERENAT case-control study. Cancer Causes Control. Apr
Barre T, Mercie P, Marcellin F, Esterle L, Duvivier C, Teicher E, Bureau M, Chas J, Salmon-Ceron D, Sogni P, Carrieri MP, Wittkop L, Protopopescu C, Group ACHS. HCV Cure and Cannabis Abstinence Facilitate Tobacco Smoking Quit Attempts in HIV-HCV Co-Infected Patients (ANRS CO13 HEPAVIH Cohort Study). AIDS Behav. Apr
Bats ML, Rucheton B, Fleur T, Orieux A, Chemin C, Rubin S, Colombies B, Desclaux A, Rivoisy C, Meriglier E,
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MASCC Abstracts
Author index
A. &Scaron MASCC
G.P. Kurita, C.N.,N.S.,H.F.,U.B.T MASCC
&# MASCC
&scaron MASCC
A, M. MASCC
A.R. MASCC
Aapro, M. MASCC
Abarca, M. MASCC
Abbasi, S. MASCC
Abbink, F.C.H. MASCC
Abbruzzese, J. MASCC
Abda, N. MASCC
Abdel Kader, Y.S. MASCC
Abdel Khalek, E.R.K. MASCC
Abdel Salam, G.A. MASCC
Abdi, F. MASCC
Abdollahi, A. MASCC
Abdolzade-Bavil, A. MASCC, MASCC
Abdul Majid, A. MASCC, MASCC
Abdul Majid, S. MASCC
Abe, H. MASCC
Abe, K. MASCC
Abella, E. MASCC, MASCC
Abernethy, A. MASCC, MASCC
Abidjonova, N. MASCC
Abimosleh, S.M. MASCC
Abisaid Baker, R. MASCC
Abraham, C. MASCC
Abrahm, J.L. MASCC
Abu, E. MASCC
Abubakar, S. MASCC
Acar, H. MASCC, MASCC, MASCC, MASCC, MASCC, MASCC, MASCC, MASCC, MASCC, MASCC, MASCC, MASCC, MASCC, MASCC, MASCC
Acevedo, A. MASCC
Achkar, S. MASCC
Ackerl, M. MASCC
Acklin, Y. MASCC
Adams, N.B. MASCC
Adamsen, L. MASC
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Published in final edited form as: Free Radic Biol Med. Jan 12;– doi: /dbiomed
Abstract
The homeostatic oxygen sensing system (HOSS) optimizes systemic oxygen delivery. Specialized tissues utilize a conserved mitochondrial sensor, often involving NDUFS2 in complex I of the mitochondrial electron transport chain, as a site of pO2-responsive production of reactive oxygen species (ROS). These ROS are converted to a diffusible signaling molecule, hydrogen peroxide (H2O2), by superoxide dismutase (SOD2). H2O2 exits the mitochondria and regulates ion channels and enzymes, altering plasma membrane potential, intracellular Ca2+ and Ca2+-sensitization and controlling acute, adaptive, responses to hypoxia that involve changes in ventilation, vascular tone and neurotransmitter release. Subversion of this O2-sensing pathway creates a pseudohypoxic state that promotes disease progression in pulmonary arterial hypertension (PAH)